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After intratracheal instillation in male Wistar rats, nickel oxide (Nio) NPs activated the NF-B pathway by upregulation of mRNA and protein expression of NF-B, an inhibitor of B kinase- and nuclear factor-inducing kinase, partially causing pulmonary damage (Chang et al., 2017)

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Ultimately, this FOXO4-DRI-mediated mechanism selectively eliminates senescent endothelial cells, which in turn attenuates vascular inflammation and improves endothelial-dependent vasodilation, conferring a significant protective effect on vascular function